The Pathophysiological Treatments of Alzheimer's Disease Literature review

The Pathophysiological Treatments of Alzheimers Disease - Literature round ExampleTau treatment has featured greatly as a means of treating the disorder across the divide. New methods like stem cell treatment and gene therapy methodologies are also under research. Their effect presently and in the future on the AD will also be mentioned. To examine the role of the research, this study aims at reviewing the pathophysiology of the AD and examines the relevant literature on the AD. It aims at creating a review that identifies the main candidate drugs and enzymes noted to play a huge role in the treatment of AD patients. It will touch on the relevant enzymes such(prenominal) as amyloid hypothesis, which play a huge role in underlining the importance of preclinical treatment for AD patients. Therapeutic modifications necessary for the treatment of AD will also be reviewed.As it is well known, Alzheimers disease (AD) is the number one cause of dementia in the world. Being one of the ne urodegenerative diseases, at that place is a need to look for ways of dealing with it. It is important to examine its pathophysiology because AD affects so many people in the world today. To tackle the start of pathophysiology, the article will look at models and mechanisms that have been operating in the general treatment of AD across the world. Beta-amyloids are the peptides of amino acids crucially problematic in the amyloid plaques in brains of patients suffering from the AD. Tau treatment involves the use of microtubule-bound protein in the treatment of AD patients. Cognitive impairment will be discussed because it is the intermediate full point that occurs as normal aging declines, based on cognitive measures. Anti-dementia drugs are important because they play a critical role in lessening the aggressiveness of AD. This will then give way to a discussion on the therapeutic targets for AD modifications. Development will only occur from a neat understanding of the present treatments and the impact they have on the degeneration of brain tissues. Many researchers suggest that